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in particular, the authors demonstrated that the absence of carnosine does not impair olfactory function in young CARNS1 −/− mice, but does in aging CARNS1 −/− mice, while there was an age-dependent decline in the number of olfactory receptor neurons in CARNS1 −/− that was not observed in wild-type mice, suggesting that carnosine is not essential for information processing in the olfactory signaling but plays a role in the long-term protection of olfactory receptor neurons

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