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Endothelial dysfunction activates NF-κB, which upregulates adhesion molecules like intercellular adhesion molecule 1 (ICAM-1) and vascular cell adhesion molecule 1 (31)
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With the breakdown of the terminal axonal cytoskeleton and mitochondrial damage 2 , activation of the complement system primarily results in membrane attack complex (MAC) production 19
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Consider whether an acceptable alternative could meet the same purpose. Avoid allowing a short deadline to erase an essential requirement.
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