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AKI triggers systemic inflammatory responses and fluid overload, which alter the permeability of the mesenteric vascular bed and contribute to intestinal edema, ultimately resulting in secondary damage to the intestinal epithelial barrier ( 3.2 Abnormal production of metabolites A growing body of evidence has confirmed that kidney diseases are associated with distinct alterations in metabolic profiles, with numerous metabolites being significantly linked to renal function decline ( In CKD, metabolic disturbances impair protein digestion and absorption, contributing to microbial dysbiosis and increased production of protein-derived metabolites, such as p-cresol, indole, phenol, and trimethylamine

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