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In AMN, VLCFAs accumulate in the spinal cord and peripheral nerves, inducing a slowly progressive dying-back axonopathy in adults, which is recognized as the core clinical syndrome of ALD [34]

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For example, in a KRas G12D ‐driven mouse pancreatic cancer model, inhibition of mTORC1 remarkably enhances the capacity of tumor cells to use extracellular protein to restore their amino acid pools, leading to increase in the tumor burden (Palm et al , 2015)