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In cultured cortical neurons from mice, MSCs inhibited NR1 and NR2A expression, which are N-Methyl-d-aspartic acid (NMDA) receptor (NMDAR) subunits that are associated with glutamate signalling (Papazian et al., 2018), and consequently, glutamate excitotoxicity was attenuated (Papazian et al., 2018)
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6a), suggesting that mitochondria are an effector of GSH restriction
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