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This process is closely linked to the activation of nuclear factor-κB (NF-κB) signaling and NOD-like receptor thermal protein domain associated protein 3 (NLRP3) inflammasomes, along with the release of pro-inflammatory cytokines, which can result in irreversible tissue damage ( in vivo and in vitro investigations have elucidated that NF-κB activation in resident glomerular cells plays a pivotal role in driving the progression of renal damage

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Takeda et al., 2001), we compared the ability of cyclin D1-Cdk6 and cyclin D1 HP mut