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To address this intellectual gap, this perspective aims to: i) introduce the mechanisms by which cytochrome P450 (CYP) enzymes function, ii) explain the physical organic phenomena that enable fluorinated analogues to avoid those processes, iii) apply those principles to rationalize the metabolic profiles of matched molecular pairs (MMPs) of fluorinated and non-fluorinated drug candidates across a variety of therapeutic classes, and importantly iv) dispel overgeneralized myths about fluorination in drug metabolism

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Factors such as hormone balance, nutrition, and even results from DNA gene testing can help tailor recommendations more precisely