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Treated with GSK3β inhibitor LiCl, the expression levels of the phosphorylated GSK3β (Ser9) was significantly upregulated, Bax and cle-Caspase3 protein were found to be significantly downregulated, and that of Bcl-2 was significantly upregulated in Prx II-knockout HaCaT cells after H 2 O 2 treatment(Fig
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